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Trofinetide

Basic information Safety Supplier Related

Trofinetide Basic information

Product Name:
Trofinetide
Synonyms:
  • Trofinetide(NNZ2566)
  • TROFINETIDE; NNZ2566 ;NNZ 2566; NNZ-2566
  • NNZ-2566
  • L-Glutamic acid, glycyl-2-methyl-L-prolyl-
  • Trofinetide
  • Trofinetide,NNZ 2566,NNZ2566,Inhibitor,inhibit
  • Glutamic acid, glycyl-​
  • prolyl-
CAS:
853400-76-7
MF:
C13H21N3O6
MW:
315.32
Product Categories:
  • API
Mol File:
853400-76-7.mol
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Trofinetide Chemical Properties

Melting point:
144 °C
Boiling point:
655.4±55.0 °C(Predicted)
Density 
1.377±0.06 g/cm3(Predicted)
storage temp. 
Store at -20°C
solubility 
Methanol (Slightly), Water (Slightly)
form 
Solid
pka
3.51±0.10(Predicted)
color 
White to Off-White
Stability:
Hygroscopic
InChI
InChI=1S/C13H21N3O6/c1-13(5-2-6-16(13)9(17)7-14)12(22)15-8(11(20)21)3-4-10(18)19/h8H,2-7,14H2,1H3,(H,15,22)(H,18,19)(H,20,21)/t8-,13-/m0/s1
InChIKey
BUSXWGRAOZQTEY-SDBXPKJASA-N
SMILES
C(O)(=O)[C@H](CCC(O)=O)NC(=O)[C@]1(C)CCCN1C(=O)CN
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Trofinetide Usage And Synthesis

Uses

Trofinetide is a synthetic analogue of the endogenous N-terminus tripeptide, Glycine-Proline-Glutamate (GPE), that has shown to be neuroprotective in animal models of brain injury.

in vivo

Trofinetide (NNZ-2566) suppresses penetrating ballistic-like brain injury (PBBI) induced inflammatory cell infiltration at 3 days following PBBI as compare to vehicle treatment. Trofinetide treatment significantly reduces the elevation of IL-6 (79%), E-selectin (81%), IL-1β (76%) and TNF-α (72%) mRNA levels in the injured hemisphere at 12 h post-PBBI, with maximal inhibition occurring between 12 h and 24 h. Trofinetide treatment does not affect the PBBI-induced up-regulation of IL-6 expression at any time point, but does produce significant reductions in the injury-induced up-regulation of IL-1β, INF-γ, and TNF-α expression. Trofinetide treatment suppresses IL-1β expression in the injured brain hemisphere for up to 7 days post-PBBI[1]. The high doses of Trofinetide (NNZ-2566) (10 and 100 mg/kg bolus followed by continuous infusion) attenuate non-convulsive seizure (NCS) occurring beyond 2 h after permanent middle cerebral artery occlusion (pMCAo). All doses of Trofinetide completely suppress the delayed occurrence of NCS as compare with the vehicle-treated animals[2].

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