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ar-turmerone

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ar-turmerone Basic information

Product Name:
ar-turmerone
Synonyms:
  • (S)-ar-Turmerone
  • TURMERONE, AR-(SH)
  • 2-Methyl-6-p-tolylhept-2-en-4-one
  • (S)-(+)-Turmerone
  • (S)-2-Methyl-6-(4-methylphenyl)-2-hepten-4-one
  • [S,(+)]-2-Methyl-6-p-tolyl-2-hepten-4-one
  • (6S)-2-methyl-6-(4-methylphenyl)hept-2-en-4-one
  • TURMERONE, AR-(SG)
CAS:
532-65-0
MF:
C15H20O
MW:
216.32
Mol File:
532-65-0.mol
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ar-turmerone Chemical Properties

Boiling point:
326℃
alpha 
D20 +82.21° (Rupe, Gassman); D22 +59.9° (c = 4.5 in hexane) (Sato)
Density 
0.945
Flash point:
122℃
storage temp. 
2-8°C
solubility 
Ethanol: Miscible
form 
Liquid
color 
Colorless to light yellow
InChI
InChI=1S/C15H20O/c1-11(2)9-15(16)10-13(4)14-7-5-12(3)6-8-14/h5-9,13H,10H2,1-4H3/t13-/m0/s1
InChIKey
NAAJVHHFAXWBOK-ZDUSSCGKSA-N
SMILES
C/C(/C)=C\C(=O)C[C@@H](C1=CC=C(C)C=C1)C
LogP
3.749 (est)
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Safety Information

Hazard Codes 
Xi,N
Risk Statements 
36-43-51/53
Safety Statements 
36/37-61
RIDADR 
UN 3082 9 / PGIII
WGK Germany 
3
HS Code 
29329990
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ar-turmerone Usage And Synthesis

Uses

(+)-ar-Turmerone-d3 is the isotope labelled analog of (+)-ar-Turmerone. (+)-ar-Turmerone is an anti-tumor and immune activating agent, which maintains a cytotoxic effect in cancer cells. Antimicrobial agent.

Definition

ChEBI: A sesquiterpenoid that is 2-methylhept-2-en-4-one substituted by a 4-methylphenyl group at position 6. It has been isolated from Peltophorum dasyrachis.

Synthesis Reference(s)

The Journal of Organic Chemistry, 38, p. 2909, 1973 DOI: 10.1021/jo00956a039
Synthetic Communications, 11, p. 579, 1981 DOI: 10.1080/00397918108063627
Tetrahedron Letters, 20, p. 1519, 1979 DOI: 10.1016/S0040-4039(01)86195-5

in vivo

ar-Turmerone (100 mg/kg; p.o.; daily for 14 days) protects hippocampal and cortical neurons by downregulating the neuroinflammatory response, thereby alleviating spatial memory impairment in mice[5].

Animal Model:Repeated intraperitoneal injections of Lipopolysaccharides (LPS) (HY-D1056) in C57BL/6 mice. [5]
Dosage:100 mg/kg
Administration:Oral gavage (p.o.); daily for 14 days
Result:Mitigated the impairment of spatial learning and reference memory in mice.
Reversed the effect of LPS induced TNF-α and IL-1βin hippocampal and cortex.
The number of normal neurons and morphological characteristics were partially restored.
Increased neuronal survival in both the hippocampus and cortex.

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