Basic information Safety Supplier Related

(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID

Basic information Safety Supplier Related

(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID Basic information

Product Name:
(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID
Synonyms:
  • dl-2-amino-4-methyl-5-phosphono-3-*pentenoic acid
  • (3E)-2-Amino-4-methyl-5-phosphono-3-pentanoic acid
  • (E)-2-Amino-4-methyl-5-phosphonopent-3-enoic acid
  • (E)-(±)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID
  • CGP 37849
  • (E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID
  • CGP 37849;CGP-37849;CGP37849
  • 3-Pentenoic acid, 2-amino-4-methyl-5-phosphono-, (3E)-
CAS:
127910-31-0
MF:
C6H12NO5P
MW:
209.14
Product Categories:
  • Glutamate receptor
  • Glutamate
Mol File:
127910-31-0.mol
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(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID Chemical Properties

Boiling point:
523.1±60.0 °C(Predicted)
Density 
1.506±0.06 g/cm3(Predicted)
storage temp. 
Desiccate at +4°C
solubility 
H2O: ≥2mg/mL (warmed)
form 
powder
pka
1.83±0.10(Predicted)
color 
white to beige
Water Solubility 
H2O: 100mM
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Safety Information

Hazard Codes 
Xi
Risk Statements 
36/37/38
Safety Statements 
26-36
WGK Germany 
3
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(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACID Usage And Synthesis

Uses

CGP 37849 is a potent antagonist at NMDA receptors which results in anticonvulsant activity (1). In addition, it may antagonize NMDA-antagonistic induced antianxiety effects (2).

Biological Activity

Potent, selective and competitive NMDA receptor antagonist (K i = 35 nM). Anticonvulsive following oral administration in vivo .

in vivo

CGP 37849 potently (Ki of 220 nM) and competitively inhibits NMDA-sensitive l-[3H]-glutamate binding to postsynaptic density (PSD) fractins from rat brain. CGP 37849 inhibits the binding of the selective NMDA receptor antagonist, [3H]-(±)-3-(2-carboxypiperazin-4-yl)propyl-1-phosphonate (CPP), with a Ki of 35 nM[1].
In vivo, oral administration to rats of CGP 37849 selectively blocks firing in hippocampal neurones induced by ionophoretically-applied NMDA, without affecting the responses to quisqualate or kainate[1].
Oral administration to mice of CGP 37849 suppresses maximal electroshock-induced seizures in mice with an ED50 of 21 mg/kg[1].

IC 50

NMDA Receptor

storage

Desiccate at +4°C

(E)-(+/-)-2-AMINO-4-METHYL-5-PHOSPHONO-3-PENTENOIC ACIDSupplier

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