Diisononyl phthalate Chemical Properties

Boiling point:

279-287 °C

Density 

0.972 g/mL at 25 °C(lit.)

Pour Point 

-54

vapor pressure 

1 mm Hg ( 200 °C)

refractive index 

n20/D 1.485(lit.)

Flash point:

>230 °F

storage temp. 

Refrigerator

solubility 

Chloroform, Methanol

form 

Oil

color 

Colourless to Pale Yellow

Merck 

13,3319

EPA Substance Registry System

Di(C8-10, C9 rich) branched alkyl phthalates (68515-48-0)

Safety Information

Risk Statements 

62-63

Safety Statements 

23-36/37

WGK Germany 

-

RTECS 

CZ3395000

TSCA 

TSCA listed

HS Code 

29173490

MSDS

• Language:English Provider:SigmaAldrich
• Language:English Provider:ACROS

Diisononyl phthalate Usage And Synthesis

Chemical Properties

The empirical formula of diisononyl phthalate (DINP) is C26H42O4. The structural formula of DINP varies because the iso-alcohols used in the manufacture contain several isomers. DINP is a clear, colorless to light yellow liquid. DINP is insoluble in water, but it is soluble in organic solvents.

Uses

Its major use is as a plasticizer.

Uses

Diisononyl Phthalate is a general-purpose plasticizer for polyvinyl chloride.

Production Methods

DINP is manufactured by the reaction of phthalic anhydride with isononanol in the presence of an acid catalyst.

Flammability and Explosibility

Non flammable

Carcinogenicity

As stated, there have been two carcinogenesis studies in F344 rats and one in B6C3F1 mice. An increased incidence of hepatocellular neoplasms (i.e., adenomas and carcinomas) is observed in both rats and mice. An increased incidence of renal cell carcinomas and mononuclear cell leukemia has also been described. These studies establish that at dose levels of approximately 600 mg/kg/day, DINP can induce hepatocellular carcinoma in rats and mice. As there was evidence of peroxisomal proliferation at the carcinogenic doses in both species, it seems most likely that this was the mechanism for hepatocellular carcinoma induction. The renal cell carcinomas observed in the male rats were associated with the induction of α 2u-globulin, indicating that it was a sex- and species-specific effect. Kidney tumors that are the consequence of α 2u-globulin induction are not considered to be clinically relevant to humans.MNCL is a tumor type that occurs spontaneously at a high and variable frequency in F344 rats. Because there is no human equivalent, MNCL is not considered to be relevant to humans.

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