MYRISTOYL-DL-CARNITINE CHLORIDE
- Product Name
- MYRISTOYL-DL-CARNITINE CHLORIDE
- CAS No.
- 14919-38-1
- Chemical Name
- MYRISTOYL-DL-CARNITINE CHLORIDE
- Synonyms
- MYRISTOYL-DL-CARNITINE CHLORIDE;(+/-)-MYRISTOYLCARNITINE CHLORIDE
- CBNumber
- CB8254739
- Molecular Formula
- C21H42ClNO4
- Formula Weight
- 408.02
- MOL File
- 14919-38-1.mol
MYRISTOYL-DL-CARNITINE CHLORIDE Property
- storage temp.
- −20°C
- solubility
- <10.2mg/ml in H2O
- form
- solid
- color
- White
- Water Solubility
- Soluble to 25 mM in water
N-Bromosuccinimide Price
- Product number
- M885003
- Product name
- (+/-)-MyristoylcarnitineChloride
- Packaging
- 500mg
- Price
- $605
- Updated
- 2021/12/16
- Product number
- B6354
- Product name
- (±)-Myristoylcarnitinechloride
- Packaging
- 50mg
- Price
- $188
- Updated
- 2021/12/16
MYRISTOYL-DL-CARNITINE CHLORIDE Chemical Properties,Usage,Production
Uses
(+/-)-Myristoylcarnitine Chloride is a homolog of Acetylcarnitine Chloride (A171990), and an intermediate in lipid metabolism.
Biological Activity
(±)-myristoylcarnitine chloride is an agonist for cholinergic and a homolog of acetylcarnitine chloride (cat no. b6273).acetylcholine receptor (achr) is an integral membrane protein receptor for acetylcholine. there are two kinds of achrs: nicotinic acetylcholine receptors and muscarinic acetylcholine receptors.(±)-myristoylcarnitine chloride is a cholinergic agonist and an intermediate in lipid metabolism [1]. in retinal ganglion cells, acetylcarnitine and acetylcholine inhibited gabaergic responses to exogenous gaba and gabaergic inhibitory postsynaptic currents [2].in dogs with coronary ligation, (-)-carnitine chloride (lcc) (300 mg/kg) and acetyl (-)-carnitine chloride (alcc) (300 mg/kg) inhibited the ventricular arrhythmia. also, lcc and alcc improved oxidative phosphorylation rate and the mitochondrial function [1]. in the mouse hot plate test, acetyl-l-carnitine (alcar) (100 mg/kg) exhibited analgesia. while, u-73122 and neomycin (the phospholipase c (plc) inhibitors) blocked the increase of the pain threshold induced by alcar. licl that impairing phosphatidylinositol synthesis antagonized the antinociception in a dose-dependent way. pma and pdbu (pkc activators) blocked the increase of the pain threshold in a dose-dependent way. these results suggested that alcar analgesia required the participation of the plc-ip3 pathway [3].
MYRISTOYL-DL-CARNITINE CHLORIDE Preparation Products And Raw materials
Raw materials
Preparation Products
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