Basic information Safety Supplier Related

L-368,899hydrochloride

Basic information Safety Supplier Related

L-368,899hydrochloride Basic information

Product Name:
L-368,899hydrochloride
Synonyms:
  • (2S)-2-Amino-N-[(1S,2S,4R)-7,7-dimethyl-1-[[[4-(2-methylphenyl)-1-piperazinyl]sulfonyl]methyl]bicyclo[2.2.1]hept-2-yl]-4-(methylsulfonyl)butanamide
  • (2S)-2-amino-N-[(1R,3S,4S)-7,7-dimethyl-4-[[4-(2-methylphenyl)piperazin-1-yl]sulfonylmethyl]-3-bicyclo[2.2.1]heptanyl]-4-methylsulfonylbutanamide
  • Butanamide, 2-amino-N-[(1S,2S,4R)-7,7-dimethyl-1-[[[4-(2-methylphenyl)-1-piperazinyl]sulfonyl]methyl]bicyclo[2.2.1]hept-2-yl]-4-(methylsulfonyl)-, (2S)-
  • L-368,899 hydrochlor
  • L-368899 free base
CAS:
148927-60-0
MF:
C26H42N4O5S2
MW:
554.77
Mol File:
148927-60-0.mol
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L-368,899hydrochloride Chemical Properties

storage temp. 
Store at +4°C
solubility 
Chloroform (Slightly), Methanol (Slightly), Water (Slightly)
form 
Solid
color 
Off-White to Pale Yellow
Stability:
Hygroscopic
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L-368,899hydrochloride Usage And Synthesis

Uses

L-368,899 Dihydrochloride is an oxytoxin antagonist and may be used in the treatment of early pregnancy failure.

Biological Activity

Potent, non-peptide and orally active oxytocin receptor antagonist (IC 50 = 8.9 nM) that displays > 40-fold selectivity over vasopressin V 1a and V 2 receptors (IC 50 values are 370 and 570 nM respectively). Antagonizes oxytocin-induced uterine contractions in vitro and in vivo .

in vivo

L-368,899 (0.1, 0.3, 1 mg/kg; infused i.v.; single) shows a dose-related antagonism of OT-stimulated uterine contractions with an AD50 value of 0.35 mg/kg in vivo[1].
L-368,899 (3, 10, 30 mg/kg; i.d.; single) inhibits the contractile effects of OT (AD50= 7 mg/kg) with a long (>4 h) duration of action in vivo (AD50: the dose of L-368,899 required to reduce the response to OT by 50%)[1].
L-368,899 (10 mg/kg, p.o.; single) shows bioavailability (AUC 0-6 h) of 35%[1].
L-368,899 (0.54, 1.8, 5.4 mg/kg; i.v.; single) reduces both oxytocin-induced and endogenous increases in plasma PGFM concentration[2].

Animal Model:Adult female Sprague-Dawley rats (250-350 g)[1].
Dosage:0.1, 0.3, 1 mg/kg
Administration:Infused intravenous injection; single.
Result:Inhibited OT-stimulated uterine contractions with an AD50 value of 0.35 mg/kg.
Animal Model:Adult female Sprague-Dawley rats (250-350 g)[1].
Dosage:3, 10, 30 mg/kg
Administration:Intraduodenal; single.
Result:Exhibited a antagonism of OT-stimulated uterine contractions with an AD50 of 7 mg/kg and duration of action more than 4 h.
Animal Model:Adult female Sprague-Dawley rats (250-350 g)[1].
Dosage:10 mg/kg
Administration:Oral administration, single.
Result:Showed orally active with bioavailability (AUC 0-6 h) of 35%.
Animal Model:Mature Dorset cross ewes (53-57 kg; Removal of ovaries)[2].
Dosage:0.54, 1.8, 5.4 mg/kg (3, 10 and 30 μg/kg/min for 3 h; dissolved in 0.9% saline).
Administration:Intravenous infusion; single.
Result:Led to a significant decrease in both the frequency (from 2.2 to 1.0 episodes/ewe) and amplitude (from 68.8 to 31.8 pg/mL) of episodes of increased plasma concentration of PGFM.

L-368,899hydrochlorideSupplier

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