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DICHLOROTRICARBONYLRUTHENIUM (II) DIMER

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DICHLOROTRICARBONYLRUTHENIUM (II) DIMER Basic information

Product Name:
DICHLOROTRICARBONYLRUTHENIUM (II) DIMER
Synonyms:
  • TRICARBONYLDICHLORORUTHENATE (II) DIMER
  • TRICARBONYLDICHLORORUTHENIUM(II) DIMER
  • DICHLOROTRICARBONYLRUTHENIUM (II) DIMER
  • Dichlorotricarbonylruthenium dimer
  • ruthenium(II) tricarbonyl chloride dimer
  • Dichlorotricarbonylruthenium(II) dimer, min. 98%
  • Dichlorotricarbonylruthenium(II) dimer,98%
  • Tetrachlorohexacarbonyldiruthenium
CAS:
22594-69-0
MF:
C6Cl4O6Ru2
MW:
512
Product Categories:
  • metal carbonyl complexes
  • Catalysis and Inorganic Chemistry
  • Ru Catalysts
  • Ruthenium
Mol File:
22594-69-0.mol
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DICHLOROTRICARBONYLRUTHENIUM (II) DIMER Chemical Properties

Melting point:
208°C (dec.)
storage temp. 
under inert gas (nitrogen or Argon) at 2-8°C
solubility 
Soluble in DMSO
form 
crystal
color 
off-white
Water Solubility 
Insoluble in water
InChIKey
JYHHJVKGDCZCCL-UHFFFAOYSA-J
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Safety Information

Hazard Codes 
Xn
Risk Statements 
20/21/22-36/37/38
Safety Statements 
26-37/39
RIDADR 
UN3466
WGK Germany 
3
HazardClass 
6.1
PackingGroup 
III

MSDS

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DICHLOROTRICARBONYLRUTHENIUM (II) DIMER Usage And Synthesis

Chemical Properties

Off-white to slightly yellow powder

Uses

Tricarbonyldichlororuthenium(II) dimer is used to enhance the coagulation and attenuation of vulnerability to fibrinolysis, modify thrombus growth or disintegration, enhances fibrinogen as a substrate for thrombin, and regulation of ion transport by gasotransmitters. Also used for various pharmacological studies. It is used as a CO donor for reactive oxygen species mediated bacterial killing.

reaction suitability

core: ruthenium
reagent type: catalyst

in vivo

Tricarbonyldichlororuthenium(II) dimer (1-10 mg/kg; oral gavage; single dose) has a protective effect in a rat model of gastric mucosal injury induced by ischemia-reperfusion[3].

Animal Model:Acute I/R gastric lesions treated male Wistar rats (250-300 g)[3]
Dosage:1, 5 or 10 mg/kg
Administration:Oral gavage (i.g.); single dose
Result:Significantly decreased the area of I/R gastric damage and significantly increased GBF at doses of 1 or 5 mg/kg.
Significantly increased mRNA expression of HMOX-1 but not HMOX-2 in gastric mucosa and significantly increased blood concentration at dose of 5 mg/kg.
Significantly increased gastric mucosal PGE2 content at dose of 5 mg/kg.
Significantly decreased mRNA expression of iNOS but not eNOS in gastric mucosa at dose of 5 mg/kg.
Significantly decreased 8-hydroxyguanozine (8-OHG) concentration in gastric mucosa at dose of 5 mg/kg.
Decreased the expression of pro- and anti-inflammatory markers' mRNA and proteins at dose of 5 mg/kg.

DICHLOROTRICARBONYLRUTHENIUM (II) DIMERSupplier

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