Basic information Safety Supplier Related

QUININE HYDROBROMIDE

Basic information Safety Supplier Related

QUININE HYDROBROMIDE Basic information

Product Name:
QUININE HYDROBROMIDE
Synonyms:
  • QUININE HYDROBROMIDE
  • 6’-methoxy-,monohydrobromide,(8-alpha,9r)-cinchonan-9-o
  • bromoquinine
  • chininhydrobromid
  • quinine,monohydrobromide
  • Quinini
  • (5-ethenyl-1-azabicyclo[2.2.2]octan-7-yl)-(6-methoxyquinolin-4-yl)methanol hydrobromide
  • (6-methoxy-4-quinolyl)-(5-vinylquinuclidin-2-yl)methanol hydrobromide
CAS:
549-49-5
MF:
C20H25BrN2O2
MW:
405.33
EINECS:
208-967-2
Mol File:
549-49-5.mol
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QUININE HYDROBROMIDE Chemical Properties

Melting point:
81-82 C
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Safety Information

Hazard Codes 
Xn,Xi
Risk Statements 
20/21/22-36/37/38-42/43
Safety Statements 
26-36
RIDADR 
1544
RTECS 
VA7650000
HazardClass 
6.1(b)
PackingGroup 
III
HS Code 
29392000
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QUININE HYDROBROMIDE Usage And Synthesis

Chemical Properties

yellow prisms

Uses

Quinidine hydrobromide is an antiarrhythmic agent. Quinidine is a potent, orally active, selective cytochrome P450db inhibitor. Quinidine hydrobromide is also a K+ channel blocker with an IC50 of 19.9 μM. Quinidine hydrobromide can be used for malaria research[1][2][3].

in vivo

Quinidine hydrobromide is rapidly absorbed, with peak plasma concentrations 60-90 min after an oral dose. Other salts (gluconate, polygalacturonate) are more slowly absorbed, with lower peak concentrations[2].
Quinidine hydrobromide is approximately 70-90 % bound to plasma proteins. It undergoes hepatic oxidative metabolism to form an N-oxide, a 3-hydroxy form, an O-demethyl form and 2'-quinidinone[2].
Quinidine hydrobromide inhibits metabolism of amphetamine in rats. Quinidine hydrobromide pretreatment results in a significant decrease in the excretion of p-hydroxyamphetamine at 24 and 48 h to 7.2 and 24.1% of the vehicle-control levels, respectively, accompanied by a significant increase in amphetamine excretion between 24 and 48 h to 542% of the control[3].

References

[1] Kehl SJ, et al. Quinidine-induced inhibition of the fast transient outward K+ current in rat melanotrophs. Br J Pharmacol. 1991 Jul;103(3):1807-13. DOI:10.1111/j.1476-5381.1991.tb09867.x
[2] Roden DM, et al. Class I antiarrhythmic agents: quinidine, procainamide and N-acetylprocainamide, disopyramide. DOI:10.1016/0163-7258(83)90012-8

QUININE HYDROBROMIDESupplier

Shanghai Qiao Chemical Science Co., Ltd
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TargetMol Chemicals Inc.
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