Basic information Safety Supplier Related

8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT

Basic information Safety Supplier Related

8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT Basic information

Product Name:
8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT
Synonyms:
  • 8-BROMOADENOSINE-3',5'-CYCLIC * MONOPHOSPHOROTHIOAT
  • 8-Bromoadenosine-3'',5''-cyclic monophosphothioate Rp isomer
  • RP-8-BR-CAMPS
  • RP-8-BR-CAMPS, NA
  • RP-8-BR-CAMPS SODIUM SALT
  • 8-bromoadenosine-3’,5’-cyclicmonophosphorothioate,rp-isomer(rp-8-br-camps),sodiumsalt
  • 8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT
  • 8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE SODIUM SALT, RPISOMER
CAS:
129735-00-8
MF:
C10H11BrN5O5PS
MW:
424.17
Mol File:
129735-00-8.mol
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8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT Chemical Properties

Boiling point:
690.3±65.0 °C(Predicted)
Density 
2.70±0.1 g/cm3(Predicted)
storage temp. 
−20°C
form 
solid
pka
-0.60±0.60(Predicted)
Water Solubility 
water: 40mg/mL
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Safety Information

Safety Statements 
22-24/25
WGK Germany 
3
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8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALT Usage And Synthesis

Uses

Rp-8-Br-cAMPS is an analog of cAMP and an inhibitor of PKA. Rp-8-Br-cAMPS occupies cAMP binding sites on PKA type I regulatory subunits, thereby preventing PKA dissociation and activation. Rp-8-Br-cAMPS can be used in the study of tumors and retrovirus-induced immune deficiency. Rp-8-Br-cAMPS also inhibits insulin secretion[1][2][3].

Biological Activity

Cell permeable: yes', 'Primary Target
PKA 1', 'Product does not compete with ATP.', 'Reversible: yes

in vivo

Rp-8-Br-cAMPS (1 mg; intraperitoneal injection; 10 days) improves immune function in a mouse retrovirus infection model[3].

Animal Model:Murine leukemia retrovirus RadLV-Rs treated male C57BL/6 mice[3]
Dosage:1 mg
Administration:Intraperitoneal injection (i.p.); 10 days
Result:Had no significant effect on the extent of lymphadenopathy and splenomegaly which is typical of RadLV-Rs retroviral infection.
Strongly increased responses to the anti-CD3 mAb.
Improved T cell responses.

References

[1] Raskovalova T, et al. Adenosine-mediated inhibition of cytotoxic activity and cytokine production by IL-2/NKp46-activated NK cells: involvement of protein kinase A isozyme I (PKA I). Immunol Res. 2006;36(1-3):91-9. DOI:10.1385/IR:36:1:91
[2] Yang S, et al. Enhanced cAMP protein kinase A signaling determines improved insulin secretion in a clonal insulin-producing beta-cell line (INS-1 832/13). Mol Endocrinol. 2004 Sep;18(9):2312-20. DOI:10.1210/me.2004-0148
[3] Nayjib B, et al. In vivo administration of a PKA type I inhibitor (Rp-8-Br-cAMPS) restores T-cell responses in retrovirus-infected mice[J]. The Open Immunology Journal, 2008, 1(1).

8-BROMOADENOSINE-3',5'-CYCLIC MONOPHOSPHOROTHIOATE, RP-ISOMER SODIUM SALTSupplier

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